the last years some interesting experimental results have been reported for experiments
in N2O, NO, NO dimer, H2, Toluene and BaFCH3 cluster. The main result consists in the observation of molecular beam
depletion when the molecules of a pulsed beam interact with a static electric
or magnetic field and an oscillating field (RF). In these cases, and as a main
difference, instead of using four fields as in the original technique developed
by I.I. Rabi and others, only two fields, those which configure the resonant
unit, are used. That is, without using the nonhomogeneous magnetic fields. The
depletion explanation for I.I. Rabi and others is based in the interaction
between the molecular electric or magnetic dipole moment and the
non-homogeneous fields. But, obviously, the change in the molecules
trajectories observed on these new experiments has to be explained without
considering the force provided by the field gradient because it happens without
using non-homogeneous fields. In this paper a theoretical way for the
explanation of these new experimental results is presented. One important point
emerges as a result of this development, namely, the existence of an, until now
unknown, spin-dependent force which would be responsible of the aforementioned
deviation of the molecules.
In this article, we prove that if B is a simple binary-Lie superalgebra whose even part is
isomorphic to sl2(F) ？and whose odd part is a completely reducible
binary-Lie-module over the even part, then B is a Lie superalgebra. We introduce also a binary-Lie
module over which is sl2(F) not completely reducible.
Our entire medical framework is based on the concept of disease, understood as a qualitative departure from normality (health) with a structural substrate (lesion), and usually an identifiable cause (aetiology). This paradigm is loaded with problems, some of which are discussed in the text. Nevertheless, we study, diagnose and treat diseases, and while often painfully conscious of the dysfunctionalities of this scheme, we can hardly imagine how we could practice medicine otherwise. However, most of the recent developments in basic sciences, and most notably in Immunology, Genetics and -omics, are inconsistent with this “health/disease” paradigm. The emerging scenario is that of complex networks, more in the spirit of Systems Biology. In these settings the qualitative difference between health and disease loses its meaning, and the whole discourse becomes progressively irreducible to our conventional clinical categories. As clinical research stagnates while basic sciences thrive, this gap is widening, and a change in the prevailing paradigm seems unavoidable. However, all our clinical judgments (including Bayesian reasoning and Evidence Based Medicine) are rooted in the disease/health dichotomy, and one can hardly conceive how they could work without it. The shift in paradigm will not be easy, and certain turmoil is to be expected.